Aryl Hydrocarbon Receptor-Deficient Mice Develop Heightened Inflammatory Responses to Cigarette Smoke and Endotoxin Associated with Rapid Loss of the Nuclear Factor-κB Component RelB

MLA

Thatcher, Thomas H., et al. “Aryl Hydrocarbon Receptor-Deficient Mice Develop Heightened Inflammatory Responses to Cigarette Smoke and Endotoxin Associated with Rapid Loss of the Nuclear Factor-ΚB Component RelB.” American Journal of Pathology, vol. 170, no. 3, Mar. 2007, pp. 855–64. EBSCOhost, https://doi.org/10.2353/ajpath.2007.060391.

APA

Thatcher, T. H., Maggirwar, S. B., Baglole, C. J., Lakatos, H. F., Gasiewicz, T. A., Phipps, R. P., & Sime, P. J. (2007). Aryl Hydrocarbon Receptor-Deficient Mice Develop Heightened Inflammatory Responses to Cigarette Smoke and Endotoxin Associated with Rapid Loss of the Nuclear Factor-κB Component RelB. American Journal of Pathology, 170(3), 855–864. https://doi.org/10.2353/ajpath.2007.060391

Chicago

Thatcher, Thomas H., Sanjay B. Maggirwar, Carolyn J. Baglole, Heather F. Lakatos, Thomas A. Gasiewicz, Richard P. Phipps, and Patricia J. Sime. 2007. “Aryl Hydrocarbon Receptor-Deficient Mice Develop Heightened Inflammatory Responses to Cigarette Smoke and Endotoxin Associated with Rapid Loss of the Nuclear Factor-ΚB Component RelB.” American Journal of Pathology 170 (3): 855–64. doi:10.2353/ajpath.2007.060391.