Neuronal overexpression of potassium channel subunit Kcnn1 prolongs survival of SOD1-linked ALS and A53T alpha-synuclein mouse models.

The article discusses a study on the overexpression of the potassium channel subunit Kcnn1 in mouse models of ALS and alpha-synuclein-related diseases. The research found that overexpression of Kcnn1 extended the survival time of the mice by up to 100% in some cases. The study suggests that the stress responses induced by the overexpressed Kcnn1 may protect against the pathogenic proteins associated with these neurodegenerative diseases. [Extracted from the article]