Receptor-interacting protein 140 overexpression impairs cardiac mitochondrial function and accelerates the transition to heart failure in chronically infarcted rats.

MLA

Chen, YanFang, et al. “Receptor-Interacting Protein 140 Overexpression Impairs Cardiac Mitochondrial Function and Accelerates the Transition to Heart Failure in Chronically Infarcted Rats.” Translational Research: The Journal of Laboratory & Clinical Medicine, vol. 180, Feb. 2017, p. 91–102.e1. EBSCOhost, https://doi.org/10.1016/j.trsl.2016.08.005.

APA

Chen, Y., Chen, S., Yue, Z., Zhang, Y., Zhou, C., Cao, W., Chen, X., Zhang, L., & Liu, P. (2017). Receptor-interacting protein 140 overexpression impairs cardiac mitochondrial function and accelerates the transition to heart failure in chronically infarcted rats. Translational Research: The Journal of Laboratory & Clinical Medicine, 180, 91–102.e1. https://doi.org/10.1016/j.trsl.2016.08.005

Chicago

Chen, YanFang, ShaoRui Chen, ZhongBao Yue, YiQiang Zhang, ChangHua Zhou, WeiWei Cao, Xi Chen, LuanKun Zhang, and PeiQing Liu. 2017. “Receptor-Interacting Protein 140 Overexpression Impairs Cardiac Mitochondrial Function and Accelerates the Transition to Heart Failure in Chronically Infarcted Rats.” Translational Research: The Journal of Laboratory & Clinical Medicine 180 (February): 91–102.e1. doi:10.1016/j.trsl.2016.08.005.