Cite
No Contribution of IP3-R(2) to Disease Phenotype in Models of Dilated Cardiomyopathy or Pressure Overload Hypertrophy.
MLA
Cooley, Nicola, et al. “No Contribution of IP3-R(2) to Disease Phenotype in Models of Dilated Cardiomyopathy or Pressure Overload Hypertrophy.” Circulation: Heart Failure, vol. 6, no. 2, Mar. 2013, pp. 318–25. EBSCOhost, https://doi.org/10.1161/CIRCHEARTFAILURE.112.972158.
APA
Cooley, N., Ouyang, K., McMullen, J. R., Kiriazis, H., Sheikh, F., Wu, W., Mu, Y., Du, X.-J., Chen, J., & Woodcock, E. A. (2013). No Contribution of IP3-R(2) to Disease Phenotype in Models of Dilated Cardiomyopathy or Pressure Overload Hypertrophy. Circulation: Heart Failure, 6(2), 318–325. https://doi.org/10.1161/CIRCHEARTFAILURE.112.972158
Chicago
Cooley, Nicola, Kunfu Ouyang, Julie R. McMullen, Helen Kiriazis, Farah Sheikh, Wei Wu, Yongxin Mu, Xiao-Jun Du, Ju Chen, and Elizabeth A. Woodcock. 2013. “No Contribution of IP3-R(2) to Disease Phenotype in Models of Dilated Cardiomyopathy or Pressure Overload Hypertrophy.” Circulation: Heart Failure 6 (2): 318–25. doi:10.1161/CIRCHEARTFAILURE.112.972158.