Administration of exogenous acylated ghrelin or rikkunshito, an endogenous ghrelin enhancer, improves the decrease in postprandial gastric motility in an acute restraint stress mouse model.

MLA

Nahata, M., et al. “Administration of Exogenous Acylated Ghrelin or Rikkunshito, an Endogenous Ghrelin Enhancer, Improves the Decrease in Postprandial Gastric Motility in an Acute Restraint Stress Mouse Model.” Neurogastroenterology & Motility, vol. 26, no. 6, June 2014, pp. 821–31. EBSCOhost, https://doi.org/10.1111/nmo.12336.

APA

Nahata, M., Saegusa, Y., Sadakane, C., Yamada, C., Nakagawa, K., Okubo, N., Ohnishi, S., Hattori, T., Sakamoto, N., & Takeda, H. (2014). Administration of exogenous acylated ghrelin or rikkunshito, an endogenous ghrelin enhancer, improves the decrease in postprandial gastric motility in an acute restraint stress mouse model. Neurogastroenterology & Motility, 26(6), 821–831. https://doi.org/10.1111/nmo.12336

Chicago

Nahata, M., Y. Saegusa, C. Sadakane, C. Yamada, K. Nakagawa, N. Okubo, S. Ohnishi, T. Hattori, N. Sakamoto, and H. Takeda. 2014. “Administration of Exogenous Acylated Ghrelin or Rikkunshito, an Endogenous Ghrelin Enhancer, Improves the Decrease in Postprandial Gastric Motility in an Acute Restraint Stress Mouse Model.” Neurogastroenterology & Motility 26 (6): 821–31. doi:10.1111/nmo.12336.