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Target protein interactions of indole-3-carbinol and the highly potent derivative 1-benzyl-I3C with the C-terminal domain of human elastase uncouples cell cycle arrest from apoptotic signaling.
- Source :
- Molecular Carcinogenesis; Nov2012, Vol. 51 Issue 11, p881-894, 14p
- Publication Year :
- 2012
Details
- Language :
- English
- ISSN :
- 08991987
- Volume :
- 51
- Issue :
- 11
- Database :
- Complementary Index
- Journal :
- Molecular Carcinogenesis
- Publication Type :
- Academic Journal
- Accession number :
- 82605521
- Full Text :
- https://doi.org/10.1002/mc.20857