Model Organisms Reveal Insight into Human Neurodegenerative Disease: Ataxin-2 Intermediate-Length Polyglutamine Expansions Are a Risk Factor for ALS.

MLA

Bonini, Nancy, and Aaron Gitler. “Model Organisms Reveal Insight into Human Neurodegenerative Disease: Ataxin-2 Intermediate-Length Polyglutamine Expansions Are a Risk Factor for ALS.” Journal of Molecular Neuroscience, vol. 45, no. 3, Nov. 2011, pp. 676–83. EBSCOhost, https://doi.org/10.1007/s12031-011-9548-9.

APA

Bonini, N., & Gitler, A. (2011). Model Organisms Reveal Insight into Human Neurodegenerative Disease: Ataxin-2 Intermediate-Length Polyglutamine Expansions Are a Risk Factor for ALS. Journal of Molecular Neuroscience, 45(3), 676–683. https://doi.org/10.1007/s12031-011-9548-9

Chicago

Bonini, Nancy, and Aaron Gitler. 2011. “Model Organisms Reveal Insight into Human Neurodegenerative Disease: Ataxin-2 Intermediate-Length Polyglutamine Expansions Are a Risk Factor for ALS.” Journal of Molecular Neuroscience 45 (3): 676–83. doi:10.1007/s12031-011-9548-9.