Cite
Involvement of NO and MEK/ERK pathway in enhancement of endothelin-1-induced mesenteric artery contraction in later-stage type 2 diabetic Goto-Kakizaki rat.
MLA
Matsumoto, Takayuki, et al. “Involvement of NO and MEK/ERK Pathway in Enhancement of Endothelin-1-Induced Mesenteric Artery Contraction in Later-Stage Type 2 Diabetic Goto-Kakizaki Rat.” American Journal of Physiology: Heart & Circulatory Physiology, vol. 296, no. 5, May 2009, pp. H1388–97. EBSCOhost, https://doi.org/10.1152/ajpheart.00043.2009.
APA
Matsumoto, T., ishida, K., Nakayama, N., Kobayashi, T., & Kamata, K. (2009). Involvement of NO and MEK/ERK pathway in enhancement of endothelin-1-induced mesenteric artery contraction in later-stage type 2 diabetic Goto-Kakizaki rat. American Journal of Physiology: Heart & Circulatory Physiology, 296(5), H1388–H1397. https://doi.org/10.1152/ajpheart.00043.2009
Chicago
Matsumoto, Takayuki, Keiko ishida, Naoaki Nakayama, Tsuneo Kobayashi, and Katsuo Kamata. 2009. “Involvement of NO and MEK/ERK Pathway in Enhancement of Endothelin-1-Induced Mesenteric Artery Contraction in Later-Stage Type 2 Diabetic Goto-Kakizaki Rat.” American Journal of Physiology: Heart & Circulatory Physiology 296 (5): H1388–97. doi:10.1152/ajpheart.00043.2009.