Colocalization of risk loci from alcoholic and novel non-alcoholic chronic pancreatitis GWAS with eQTL signals indicates candidate disease causing mechanisms.

MLA

Schmidt, Andreas W., et al. “Colocalization of Risk Loci from Alcoholic and Novel Non-Alcoholic Chronic Pancreatitis GWAS with EQTL Signals Indicates Candidate Disease Causing Mechanisms.” Pancreatology (Elsevier Science), vol. 22, Nov. 2022, p. e40. EBSCOhost, https://doi.org/10.1016/j.pan.2022.06.110.

APA

Schmidt, A. W., Kühnapfel, A., Kirsten, H., Witt, H., Laumen, H., & Rosendahl, J. (2022). Colocalization of risk loci from alcoholic and novel non-alcoholic chronic pancreatitis GWAS with eQTL signals indicates candidate disease causing mechanisms. Pancreatology (Elsevier Science), 22, e40. https://doi.org/10.1016/j.pan.2022.06.110

Chicago

Schmidt, Andreas W., A. Kühnapfel, H. Kirsten, H. Witt, H. Laumen, and J. Rosendahl. 2022. “Colocalization of Risk Loci from Alcoholic and Novel Non-Alcoholic Chronic Pancreatitis GWAS with EQTL Signals Indicates Candidate Disease Causing Mechanisms.” Pancreatology (Elsevier Science) 22 (November): e40. doi:10.1016/j.pan.2022.06.110.