High replication stress and limited Rad51-mediated DNA repair capacity, but not oxidative stress, underlie oligodendrocyte precursor cell radiosensitivity.

MLA

Berger, N.Daniel, et al. “High Replication Stress and Limited Rad51-Mediated DNA Repair Capacity, but Not Oxidative Stress, Underlie Oligodendrocyte Precursor Cell Radiosensitivity.” NAR Cancer, vol. 4, no. 2, June 2022, pp. 1–18. EBSCOhost, https://doi.org/10.1093/narcan/zcac012.

APA

Berger, N. D., Brownlee, P. M., Chen, M. J., Morrison, H., Osz, K., Ploquin, N. P., Chan, J. A., & Goodarzi, A. A. (2022). High replication stress and limited Rad51-mediated DNA repair capacity, but not oxidative stress, underlie oligodendrocyte precursor cell radiosensitivity. NAR Cancer, 4(2), 1–18. https://doi.org/10.1093/narcan/zcac012

Chicago

Berger, N. Daniel, Peter M. Brownlee, Myra J. Chen, Hali Morrison, Katalin Osz, Nicolas P. Ploquin, Jennifer A. Chan, and Aaron A. Goodarzi. 2022. “High Replication Stress and Limited Rad51-Mediated DNA Repair Capacity, but Not Oxidative Stress, Underlie Oligodendrocyte Precursor Cell Radiosensitivity.” NAR Cancer 4 (2): 1–18. doi:10.1093/narcan/zcac012.