Intracellular acidosis-activated p38 MAPK signaling and its essential role in cardiomyocyte hypoxic injury.

Presents a study that investigated the possible effect of intracellular acidosis, a characteristic of hypoxic and ischemic myocardium, on p38 mitogen-activated protein kinase signaling and its pathophysiological relevance in myocardium hypoxia. Principal findings; Conclusions; Significance.