Cite
Sialomucin CD43 Plays a Deleterious Role in the Development of Experimental Heart Failure Induced by Pressure Overload by Modulating Cardiac Inflammation and Fibrosis.
MLA
Kaur, Kuljeet, et al. “Sialomucin CD43 Plays a Deleterious Role in the Development of Experimental Heart Failure Induced by Pressure Overload by Modulating Cardiac Inflammation and Fibrosis.” Frontiers in Physiology, vol. 12, Dec. 2021, pp. 1–10. EBSCOhost, https://doi.org/10.3389/fphys.2021.780854.
APA
Kaur, K., Velázquez, F. E., Anastasiou, M., Ngwenyama, N., Smolgovsky, S., Aronovitz, M., & Alcaide, P. (2021). Sialomucin CD43 Plays a Deleterious Role in the Development of Experimental Heart Failure Induced by Pressure Overload by Modulating Cardiac Inflammation and Fibrosis. Frontiers in Physiology, 12, 1–10. https://doi.org/10.3389/fphys.2021.780854
Chicago
Kaur, Kuljeet, Francisco E. Velázquez, Marina Anastasiou, Njabulo Ngwenyama, Sasha Smolgovsky, Mark Aronovitz, and Pilar Alcaide. 2021. “Sialomucin CD43 Plays a Deleterious Role in the Development of Experimental Heart Failure Induced by Pressure Overload by Modulating Cardiac Inflammation and Fibrosis.” Frontiers in Physiology 12 (December): 1–10. doi:10.3389/fphys.2021.780854.