Substitution of conserved glycine residue by alanine in natural and synthetic neuropeptide ligands causes partial agonism at the stomoxytachykinin receptor.

MLA

Poels, Jeroen, et al. “Substitution of Conserved Glycine Residue by Alanine in Natural and Synthetic Neuropeptide Ligands Causes Partial Agonism at the Stomoxytachykinin Receptor.” Journal of Neurochemistry, vol. 90, no. 2, July 2004, pp. 472–78. EBSCOhost, https://doi.org/10.1111/j.1471-4159.2004.02506.x.

APA

Poels, J., van Loy, T., Franssens, V., Detheux, M., Nachman, R. J., Oonk, H. B., Åkerman, K. E., Vassart, G., Parmentier, M., de Loof, A., Torfs, H., & vanden Broeck, J. (2004). Substitution of conserved glycine residue by alanine in natural and synthetic neuropeptide ligands causes partial agonism at the stomoxytachykinin receptor. Journal of Neurochemistry, 90(2), 472–478. https://doi.org/10.1111/j.1471-4159.2004.02506.x

Chicago

Poels, Jeroen, Tom van Loy, Vanessa Franssens, Michel Detheux, Ronald J. Nachman, Hendrica B. Oonk, Karl E. Åkerman, et al. 2004. “Substitution of Conserved Glycine Residue by Alanine in Natural and Synthetic Neuropeptide Ligands Causes Partial Agonism at the Stomoxytachykinin Receptor.” Journal of Neurochemistry 90 (2): 472–78. doi:10.1111/j.1471-4159.2004.02506.x.