A critical role of PI-3K/Akt/JNKs pathway in benzo[a]pyrene diol-epoxide (B[a]PDE)-induced AP-1 transactivation in mouse epidermal Cl41 cells.

MLA

Li, Jingxia, et al. “A Critical Role of PI-3K/Akt/JNKs Pathway in Benzo[a]Pyrene Diol-Epoxide (B[a]PDE)-Induced AP-1 Transactivation in Mouse Epidermal Cl41 Cells.” Oncogene, vol. 23, no. 22, May 2004, pp. 3932–44. EBSCOhost, https://doi.org/10.1038/sj.onc.1207501.

APA

Li, J., Tang, M., Liu, B., Shi, X., & Huang, C. (2004). A critical role of PI-3K/Akt/JNKs pathway in benzo[a]pyrene diol-epoxide (B[a]PDE)-induced AP-1 transactivation in mouse epidermal Cl41 cells. Oncogene, 23(22), 3932–3944. https://doi.org/10.1038/sj.onc.1207501

Chicago

Li, Jingxia, Moon-shong Tang, Bingci Liu, Xianglin Shi, and Chuanshu Huang. 2004. “A Critical Role of PI-3K/Akt/JNKs Pathway in Benzo[a]Pyrene Diol-Epoxide (B[a]PDE)-Induced AP-1 Transactivation in Mouse Epidermal Cl41 Cells.” Oncogene 23 (22): 3932–44. doi:10.1038/sj.onc.1207501.