A bioinformatic and mechanistic study elicits the antifibrotic effect of ursolic acid through the attenuation of oxidative stress with the involvement of ERK, PI3K/Akt, and p38 MAPK signaling pathways in human hepatic stellate cells and rat liver.

MLA

Wenhua He, et al. “A Bioinformatic and Mechanistic Study Elicits the Antifibrotic Effect of Ursolic Acid through the Attenuation of Oxidative Stress with the Involvement of ERK, PI3K/Akt, and P38 MAPK Signaling Pathways in Human Hepatic Stellate Cells and Rat Liver.” Drug Design, Development & Therapy, vol. 9, July 2015, pp. 3989–4104. EBSCOhost, https://doi.org/10.2147/DDDT.S85426.

APA

Wenhua He, Feng Shi, Zhi-Wei Zhou, Bimin Li, Kunhe Zhang, Xinhua Zhang, Canhui Ouyang, Shu-Feng Zhou, & Xuan Zhu. (2015). A bioinformatic and mechanistic study elicits the antifibrotic effect of ursolic acid through the attenuation of oxidative stress with the involvement of ERK, PI3K/Akt, and p38 MAPK signaling pathways in human hepatic stellate cells and rat liver. Drug Design, Development & Therapy, 9, 3989–4104. https://doi.org/10.2147/DDDT.S85426

Chicago

Wenhua He, Feng Shi, Zhi-Wei Zhou, Bimin Li, Kunhe Zhang, Xinhua Zhang, Canhui Ouyang, Shu-Feng Zhou, and Xuan Zhu. 2015. “A Bioinformatic and Mechanistic Study Elicits the Antifibrotic Effect of Ursolic Acid through the Attenuation of Oxidative Stress with the Involvement of ERK, PI3K/Akt, and P38 MAPK Signaling Pathways in Human Hepatic Stellate Cells and Rat Liver.” Drug Design, Development & Therapy 9 (July): 3989–4104. doi:10.2147/DDDT.S85426.