Cite
Increased intestinal permeability and tight junction disruption by altered expression and localization of occludin in a murine graft versus host disease model.
MLA
Noth, Rainer, et al. “Increased Intestinal Permeability and Tight Junction Disruption by Altered Expression and Localization of Occludin in a Murine Graft versus Host Disease Model.” BMC Gastroenterology, vol. 11, no. 1, Jan. 2011, p. 109. EBSCOhost, https://doi.org/10.1186/1471-230X-11-109.
APA
Noth, R., Lange-Grumfeld, J., Stüber, E., Kruse, M.-L., Ellrichmann, M., Häsler, R., Hampe, J., Bewig, B., Rosenstiel, P., Schreiber, S., & Arlt, A. (2011). Increased intestinal permeability and tight junction disruption by altered expression and localization of occludin in a murine graft versus host disease model. BMC Gastroenterology, 11(1), 109. https://doi.org/10.1186/1471-230X-11-109
Chicago
Noth, Rainer, Julia Lange-Grumfeld, Eckhard Stüber, Marie-Luise Kruse, Mark Ellrichmann, Robert Häsler, Jochen Hampe, et al. 2011. “Increased Intestinal Permeability and Tight Junction Disruption by Altered Expression and Localization of Occludin in a Murine Graft versus Host Disease Model.” BMC Gastroenterology 11 (1): 109. doi:10.1186/1471-230X-11-109.