Interleukin-23 Mediates the Intestinal Response to Microbial β-1,3-Glucan and the Development of Spondyloarthritis Pathology in SKG Mice.

MLA

Benham, Helen, et al. “Interleukin-23 Mediates the Intestinal Response to Microbial β-1,3-Glucan and the Development of Spondyloarthritis Pathology in SKG Mice.” Arthritis & Rheumatology, vol. 66, no. 7, July 2014, pp. 1755–67. EBSCOhost, https://doi.org/10.1002/art.38638.

APA

Benham, H., Rehaume, L. M., Hasnain, S. Z., Velasco, J., Baillet, A. C., Ruutu, M., Kikly, K., Wang, R., Tseng, H., Thomas, G. P., Brown, M. A., Strutton, G., McGuckin, M. A., & Thomas, R. (2014). Interleukin-23 Mediates the Intestinal Response to Microbial β-1,3-Glucan and the Development of Spondyloarthritis Pathology in SKG Mice. Arthritis & Rheumatology, 66(7), 1755–1767. https://doi.org/10.1002/art.38638

Chicago

Benham, Helen, Linda M. Rehaume, Sumaira Z. Hasnain, Jared Velasco, Athan C. Baillet, Merja Ruutu, Kristine Kikly, et al. 2014. “Interleukin-23 Mediates the Intestinal Response to Microbial β-1,3-Glucan and the Development of Spondyloarthritis Pathology in SKG Mice.” Arthritis & Rheumatology 66 (7): 1755–67. doi:10.1002/art.38638.