Glycylcyclines bind to the high-affinity tetracycline ribosomal binding site and evade Tet(M)- and Tet(O)-mediated ribosomal protection.

MLA

Bergeron, J., et al. “Glycylcyclines Bind to the High-Affinity Tetracycline Ribosomal Binding Site and Evade Tet(M)- and Tet(O)-Mediated Ribosomal Protection.” Antimicrobial Agents and Chemotherapy, vol. 40, no. 9, Sept. 1996, pp. 2226–28. EBSCOhost, https://doi.org/10.1128/AAC.40.9.2226.

APA

Bergeron, J., Ammirati, M., Danley, D., James, L., Norcia, M., Retsema, J., Strick, C. A., Su, W. G., Sutcliffe, J., & Wondrack, L. (1996). Glycylcyclines bind to the high-affinity tetracycline ribosomal binding site and evade Tet(M)- and Tet(O)-mediated ribosomal protection. Antimicrobial Agents and Chemotherapy, 40(9), 2226–2228. https://doi.org/10.1128/AAC.40.9.2226

Chicago

Bergeron, J, M Ammirati, D Danley, L James, M Norcia, J Retsema, C A Strick, W G Su, J Sutcliffe, and L Wondrack. 1996. “Glycylcyclines Bind to the High-Affinity Tetracycline Ribosomal Binding Site and Evade Tet(M)- and Tet(O)-Mediated Ribosomal Protection.” Antimicrobial Agents and Chemotherapy 40 (9): 2226–28. doi:10.1128/AAC.40.9.2226.