Tendon hypertrophy is associated with increased hydroxylation of nonhelical lysine residues at two specific cross-linking sites in type I collagen.

MLA

Gerriets, J. E., et al. “Tendon Hypertrophy Is Associated with Increased Hydroxylation of Nonhelical Lysine Residues at Two Specific Cross-Linking Sites in Type I Collagen.” The Journal of Biological Chemistry, vol. 268, no. 34, Dec. 1993, pp. 25553–60. EBSCOhost, widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=cmedm&AN=8244992&authtype=sso&custid=ns315887.

APA

Gerriets, J. E., Curwin, S. L., & Last, J. A. (1993). Tendon hypertrophy is associated with increased hydroxylation of nonhelical lysine residues at two specific cross-linking sites in type I collagen. The Journal of Biological Chemistry, 268(34), 25553–25560.

Chicago

Gerriets, J E, S L Curwin, and J A Last. 1993. “Tendon Hypertrophy Is Associated with Increased Hydroxylation of Nonhelical Lysine Residues at Two Specific Cross-Linking Sites in Type I Collagen.” The Journal of Biological Chemistry 268 (34): 25553–60. http://widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=cmedm&AN=8244992&authtype=sso&custid=ns315887.