Phenobarbital-mediated increase in ring- and N-hydroxylation of the carcinogen N-2-fluorenylacetamide, and decrease in amounts bound to liver deoxyribonucleic acid.

MLA

Matsushima, T., et al. “Phenobarbital-Mediated Increase in Ring- and N-Hydroxylation of the Carcinogen N-2-Fluorenylacetamide, and Decrease in Amounts Bound to Liver Deoxyribonucleic Acid.” Biochemical Pharmacology, vol. 21, no. 15, Aug. 1972, pp. 2043–51. EBSCOhost, https://doi.org/10.1016/0006-2952(72)90158-x.

APA

Matsushima, T., Grantham, P. H., Weisburger, E. K., & Weisburger, J. H. (1972). Phenobarbital-mediated increase in ring- and N-hydroxylation of the carcinogen N-2-fluorenylacetamide, and decrease in amounts bound to liver deoxyribonucleic acid. Biochemical Pharmacology, 21(15), 2043–2051. https://doi.org/10.1016/0006-2952(72)90158-x

Chicago

Matsushima, T, P H Grantham, E K Weisburger, and J H Weisburger. 1972. “Phenobarbital-Mediated Increase in Ring- and N-Hydroxylation of the Carcinogen N-2-Fluorenylacetamide, and Decrease in Amounts Bound to Liver Deoxyribonucleic Acid.” Biochemical Pharmacology 21 (15): 2043–51. doi:10.1016/0006-2952(72)90158-x.