Integrated multiomic analysis identifies TRIP13 as a mediator of alveolar epithelial type II cell dysfunction in idiopathic pulmonary fibrosis.

MLA

St Pierre, Laurence, et al. “Integrated Multiomic Analysis Identifies TRIP13 as a Mediator of Alveolar Epithelial Type II Cell Dysfunction in Idiopathic Pulmonary Fibrosis.” Biochimica et Biophysica Acta. Molecular Basis of Disease, vol. 1871, no. 3, Nov. 2024, p. 167572. EBSCOhost, https://doi.org/10.1016/j.bbadis.2024.167572.

APA

St Pierre, L., Berhan, A., Sung, E. K., Alvarez, J. R., Wang, H., Ji, Y., Liu, Y., Yu, H., Meier, A., Afshar, K., Golts, E. M., Lin, G. Y., Castaldi, A., Calvert, B. A., Ryan, A., Zhou, B., Offringa, I. A., Marconett, C. N., & Borok, Z. (2024). Integrated multiomic analysis identifies TRIP13 as a mediator of alveolar epithelial type II cell dysfunction in idiopathic pulmonary fibrosis. Biochimica et Biophysica Acta. Molecular Basis of Disease, 1871(3), 167572. https://doi.org/10.1016/j.bbadis.2024.167572

Chicago

St Pierre, Laurence, Asres Berhan, Eun K Sung, Juan R Alvarez, Hongjun Wang, Yanbin Ji, Yixin Liu, et al. 2024. “Integrated Multiomic Analysis Identifies TRIP13 as a Mediator of Alveolar Epithelial Type II Cell Dysfunction in Idiopathic Pulmonary Fibrosis.” Biochimica et Biophysica Acta. Molecular Basis of Disease 1871 (3): 167572. doi:10.1016/j.bbadis.2024.167572.