Prenatal exposure to di-n-butyl phthalate promotes RIPK1-regulated necroptosis of uroepithelial cells and induces hypospadias through the epithelial-mesenchymal transition process in newborn male rats.

MLA

Wu, Lei, et al. “Prenatal Exposure to Di-n-Butyl Phthalate Promotes RIPK1-Regulated Necroptosis of Uroepithelial Cells and Induces Hypospadias through the Epithelial-Mesenchymal Transition Process in Newborn Male Rats.” Toxicology, vol. 509, Dec. 2024, p. 153982. EBSCOhost, https://doi.org/10.1016/j.tox.2024.153982.

APA

Wu, L., Xie, Z., Li, T., Chen, X., Jiang, J., Shi, F., Zhang, Y., Xu, X., Xia, S., & Sun, W. (2024). Prenatal exposure to di-n-butyl phthalate promotes RIPK1-regulated necroptosis of uroepithelial cells and induces hypospadias through the epithelial-mesenchymal transition process in newborn male rats. Toxicology, 509, 153982. https://doi.org/10.1016/j.tox.2024.153982

Chicago

Wu, Lei, Zhiwen Xie, Tiewen Li, Xincan Chen, Juntao Jiang, Fei Shi, Yongqing Zhang, Xinyu Xu, Shujie Xia, and Wenlan Sun. 2024. “Prenatal Exposure to Di-n-Butyl Phthalate Promotes RIPK1-Regulated Necroptosis of Uroepithelial Cells and Induces Hypospadias through the Epithelial-Mesenchymal Transition Process in Newborn Male Rats.” Toxicology 509 (December): 153982. doi:10.1016/j.tox.2024.153982.