Cite
Salinomycin, a potent inhibitor of XOD and URAT1, ameliorates hyperuricemic nephropathy by activating NRF2, modulating the gut microbiota, and promoting SCFA production.
MLA
Chen, Yong-Jun, et al. “Salinomycin, a Potent Inhibitor of XOD and URAT1, Ameliorates Hyperuricemic Nephropathy by Activating NRF2, Modulating the Gut Microbiota, and Promoting SCFA Production.” Chemico-Biological Interactions, vol. 403, Nov. 2024, p. 111220. EBSCOhost, https://doi.org/10.1016/j.cbi.2024.111220.
APA
Chen, Y.-J., Guo, Z.-T., Chen, H.-Q., Zhang, S.-F., Bao, Y.-X., Xie, Z., Ke, J., Ye, W.-J., Liang, J.-C., Chen, J.-C., Li, N., Zheng, F.-X., Liao, H., Wu, T., & Pang, J.-X. (2024). Salinomycin, a potent inhibitor of XOD and URAT1, ameliorates hyperuricemic nephropathy by activating NRF2, modulating the gut microbiota, and promoting SCFA production. Chemico-Biological Interactions, 403, 111220. https://doi.org/10.1016/j.cbi.2024.111220
Chicago
Chen, Yong-Jun, Zi-Tao Guo, Hai-Qiao Chen, Shi-Fan Zhang, Ying-Xia Bao, Zhoufan Xie, Jia-le Ke, et al. 2024. “Salinomycin, a Potent Inhibitor of XOD and URAT1, Ameliorates Hyperuricemic Nephropathy by Activating NRF2, Modulating the Gut Microbiota, and Promoting SCFA Production.” Chemico-Biological Interactions 403 (November): 111220. doi:10.1016/j.cbi.2024.111220.