Cellular prion protein acts as mediator of amyloid beta uptake by caveolin-1 causing cellular dysfunctions in vitro and in vivo.

MLA

da Silva Correia, Angela, et al. “Cellular Prion Protein Acts as Mediator of Amyloid Beta Uptake by Caveolin-1 Causing Cellular Dysfunctions in Vitro and in Vivo.” Alzheimer’s & Dementia : The Journal of the Alzheimer’s Association, vol. 20, no. 10, Oct. 2024, pp. 6776–92. EBSCOhost, https://doi.org/10.1002/alz.14120.

APA

da Silva Correia, A., Schmitz, M., Fischer, A.-L., da Silva Correia, S., Simonetti, F. L., Saher, G., Goya-Maldonado, R., Arora, A. S., Fischer, A., Outeiro, T. F., & Zerr, I. (2024). Cellular prion protein acts as mediator of amyloid beta uptake by caveolin-1 causing cellular dysfunctions in vitro and in vivo. Alzheimer’s & Dementia : The Journal of the Alzheimer’s Association, 20(10), 6776–6792. https://doi.org/10.1002/alz.14120

Chicago

da Silva Correia, Angela, Matthias Schmitz, Anna-Lisa Fischer, Susana da Silva Correia, Franco L Simonetti, Gesine Saher, Roberto Goya-Maldonado, et al. 2024. “Cellular Prion Protein Acts as Mediator of Amyloid Beta Uptake by Caveolin-1 Causing Cellular Dysfunctions in Vitro and in Vivo.” Alzheimer’s & Dementia : The Journal of the Alzheimer’s Association 20 (10): 6776–92. doi:10.1002/alz.14120.