The crosstalk between oxidative stress and DNA damage induces neural stem cell senescence by HO-1/PARP1 non-canonical pathway.

MLA

Li, Cheng, et al. “The Crosstalk between Oxidative Stress and DNA Damage Induces Neural Stem Cell Senescence by HO-1/PARP1 Non-Canonical Pathway.” Free Radical Biology & Medicine, vol. 223, Oct. 2024, pp. 443–57. EBSCOhost, https://doi.org/10.1016/j.freeradbiomed.2024.07.020.

APA

Li, C., Wu, J., Dong, Q., Ma, J., Gao, H., Liu, G., Chen, Y., Ning, J., Lv, X., Zhang, M., Zhong, H., Zheng, T., Liu, Y., Peng, Y., Qu, Y., Gao, X., Shi, H., Sun, C., & Hui, Y. (2024). The crosstalk between oxidative stress and DNA damage induces neural stem cell senescence by HO-1/PARP1 non-canonical pathway. Free Radical Biology & Medicine, 223, 443–457. https://doi.org/10.1016/j.freeradbiomed.2024.07.020

Chicago

Li, Cheng, Jiajia Wu, Qi Dong, Jiajia Ma, Huiqun Gao, Guiyan Liu, You Chen, et al. 2024. “The Crosstalk between Oxidative Stress and DNA Damage Induces Neural Stem Cell Senescence by HO-1/PARP1 Non-Canonical Pathway.” Free Radical Biology & Medicine 223 (October): 443–57. doi:10.1016/j.freeradbiomed.2024.07.020.