Inactivated cGAS-STING Signaling Facilitates Endocrine Resistance by Forming a Positive Feedback Loop with AKT Kinase in ER+HER2- Breast Cancer.

MLA

Zhang, Kai-Ming, et al. “Inactivated CGAS-STING Signaling Facilitates Endocrine Resistance by Forming a Positive Feedback Loop with AKT Kinase in ER+HER2- Breast Cancer.” Advanced Science (Weinheim, Baden-Wurttemberg, Germany), vol. 11, no. 35, Sept. 2024, p. e2403592. EBSCOhost, https://doi.org/10.1002/advs.202403592.

APA

Zhang, K.-M., Zhao, D.-C., Li, Z.-Y., Wang, Y., Liu, J.-N., Du, T., Zhou, L., Chen, Y.-H., Yu, Q.-C., Chen, Q.-S., Cai, R.-Z., Zhao, Z.-X., Shan, J.-L., Hu, B.-X., Zhang, H.-L., Feng, G.-K., Zhu, X.-F., Tang, J., & Deng, R. (2024). Inactivated cGAS-STING Signaling Facilitates Endocrine Resistance by Forming a Positive Feedback Loop with AKT Kinase in ER+HER2- Breast Cancer. Advanced Science (Weinheim, Baden-Wurttemberg, Germany), 11(35), e2403592. https://doi.org/10.1002/advs.202403592

Chicago

Zhang, Kai-Ming, De-Chang Zhao, Ze-Yu Li, Yan Wang, Jian-Nan Liu, Tian Du, Ling Zhou, et al. 2024. “Inactivated CGAS-STING Signaling Facilitates Endocrine Resistance by Forming a Positive Feedback Loop with AKT Kinase in ER+HER2- Breast Cancer.” Advanced Science (Weinheim, Baden-Wurttemberg, Germany) 11 (35): e2403592. doi:10.1002/advs.202403592.