Increased PHLPP1 expression through ERK-4E-BP1 signaling axis drives nicotine induced oxidative stress related damage of cardiomyocytes.

MLA

Mohammed Abdul, Khaja Shameem, et al. “Increased PHLPP1 Expression through ERK-4E-BP1 Signaling Axis Drives Nicotine Induced Oxidative Stress Related Damage of Cardiomyocytes.” Journal of Molecular and Cellular Cardiology, vol. 193, Aug. 2024, pp. 100–12. EBSCOhost, https://doi.org/10.1016/j.yjmcc.2024.05.014.

APA

Mohammed Abdul, K. S., Han, K., Guerrero, A. B., Wilson, C. N., Kulkarni, A., & Purcell, N. H. (2024). Increased PHLPP1 expression through ERK-4E-BP1 signaling axis drives nicotine induced oxidative stress related damage of cardiomyocytes. Journal of Molecular and Cellular Cardiology, 193, 100–112. https://doi.org/10.1016/j.yjmcc.2024.05.014

Chicago

Mohammed Abdul, Khaja Shameem, Kimin Han, Alyssa B Guerrero, Cekia N Wilson, Amogh Kulkarni, and Nicole H Purcell. 2024. “Increased PHLPP1 Expression through ERK-4E-BP1 Signaling Axis Drives Nicotine Induced Oxidative Stress Related Damage of Cardiomyocytes.” Journal of Molecular and Cellular Cardiology 193 (August): 100–112. doi:10.1016/j.yjmcc.2024.05.014.