Mitochondrial transplantation attenuates oligomeric amyloid-beta-induced mitochondrial dysfunction and tight junction protein destruction in retinal pigment epithelium.

MLA

Noh, Sung-Eun, et al. “Mitochondrial Transplantation Attenuates Oligomeric Amyloid-Beta-Induced Mitochondrial Dysfunction and Tight Junction Protein Destruction in Retinal Pigment Epithelium.” Free Radical Biology & Medicine, vol. 212, Feb. 2024, pp. 10–21. EBSCOhost, https://doi.org/10.1016/j.freeradbiomed.2023.12.012.

APA

Noh, S.-E., Lee, S. J., Cho, C. S., Jo, D. H., Park, K. S., & Kim, J. H. (2024). Mitochondrial transplantation attenuates oligomeric amyloid-beta-induced mitochondrial dysfunction and tight junction protein destruction in retinal pigment epithelium. Free Radical Biology & Medicine, 212, 10–21. https://doi.org/10.1016/j.freeradbiomed.2023.12.012

Chicago

Noh, Sung-Eun, Seok Jae Lee, Chang Sik Cho, Dong Hyun Jo, Kyu Sang Park, and Jeong Hun Kim. 2024. “Mitochondrial Transplantation Attenuates Oligomeric Amyloid-Beta-Induced Mitochondrial Dysfunction and Tight Junction Protein Destruction in Retinal Pigment Epithelium.” Free Radical Biology & Medicine 212 (February): 10–21. doi:10.1016/j.freeradbiomed.2023.12.012.