Cite
Synaptic remodeling of GluA1 and GluA2 expression in the nucleus accumbens promotes susceptibility to cognitive deficits concomitant with downstream GSK3 β mediated neurotoxicity in female mice during abstinence from voluntary oral methamphetamine.
MLA
Memos, Nicoletta, et al. “Synaptic Remodeling of GluA1 and GluA2 Expression in the Nucleus Accumbens Promotes Susceptibility to Cognitive Deficits Concomitant with Downstream GSK3 β Mediated Neurotoxicity in Female Mice during Abstinence from Voluntary Oral Methamphetamine.” Addiction Neuroscience, vol. 8, Dec. 2023. EBSCOhost, https://doi.org/10.1016/j.addicn.2023.100112.
APA
Memos, N., Avila, J. A., Rodriguez, E., & Serrano, P. A. (2023). Synaptic remodeling of GluA1 and GluA2 expression in the nucleus accumbens promotes susceptibility to cognitive deficits concomitant with downstream GSK3 β mediated neurotoxicity in female mice during abstinence from voluntary oral methamphetamine. Addiction Neuroscience, 8. https://doi.org/10.1016/j.addicn.2023.100112
Chicago
Memos, Nicoletta, Jorge A Avila, Edgar Rodriguez, and Peter A Serrano. 2023. “Synaptic Remodeling of GluA1 and GluA2 Expression in the Nucleus Accumbens Promotes Susceptibility to Cognitive Deficits Concomitant with Downstream GSK3 β Mediated Neurotoxicity in Female Mice during Abstinence from Voluntary Oral Methamphetamine.” Addiction Neuroscience 8 (December). doi:10.1016/j.addicn.2023.100112.