GPR109A controls neutrophil extracellular traps formation and improve early sepsis by regulating ROS/PAD4/Cit-H3 signal axis.

MLA

Guo, Wenjin, et al. “GPR109A Controls Neutrophil Extracellular Traps Formation and Improve Early Sepsis by Regulating ROS/PAD4/Cit-H3 Signal Axis.” Experimental Hematology & Oncology, vol. 12, no. 1, Jan. 2023, p. 15. EBSCOhost, https://doi.org/10.1186/s40164-023-00376-4.

APA

Guo, W., Gong, Q., Zong, X., Wu, D., Li, Y., Xiao, H., Song, J., Zhang, S., Fu, S., Feng, Z., & Zhuang, L. (2023). GPR109A controls neutrophil extracellular traps formation and improve early sepsis by regulating ROS/PAD4/Cit-H3 signal axis. Experimental Hematology & Oncology, 12(1), 15. https://doi.org/10.1186/s40164-023-00376-4

Chicago

Guo, Wenjin, Qian Gong, Xiaofeng Zong, Dianjun Wu, Yuhang Li, Huijie Xiao, Jie Song, et al. 2023. “GPR109A Controls Neutrophil Extracellular Traps Formation and Improve Early Sepsis by Regulating ROS/PAD4/Cit-H3 Signal Axis.” Experimental Hematology & Oncology 12 (1): 15. doi:10.1186/s40164-023-00376-4.