A new mouse model of post-traumatic joint injury allows to identify the contribution of Gli1+ mesenchymal progenitors in arthrofibrosis and acquired heterotopic endochondral ossification.

MLA

Magallanes, Jenny, et al. “A New Mouse Model of Post-Traumatic Joint Injury Allows to Identify the Contribution of Gli1+ Mesenchymal Progenitors in Arthrofibrosis and Acquired Heterotopic Endochondral Ossification.” Frontiers in Cell and Developmental Biology, vol. 10, Aug. 2022, p. 954028. EBSCOhost, https://doi.org/10.3389/fcell.2022.954028.

APA

Magallanes, J., Liu, N. Q., Zhang, J., Ouyang, Y., Mkaratigwa, T., Bian, F., Van Handel, B., Skorka, T., Petrigliano, F. A., & Evseenko, D. (2022). A new mouse model of post-traumatic joint injury allows to identify the contribution of Gli1+ mesenchymal progenitors in arthrofibrosis and acquired heterotopic endochondral ossification. Frontiers in Cell and Developmental Biology, 10, 954028. https://doi.org/10.3389/fcell.2022.954028

Chicago

Magallanes, Jenny, Nancy Q Liu, Jiankang Zhang, Yuxin Ouyang, Tadiwanashe Mkaratigwa, Fangzhou Bian, Ben Van Handel, Tautis Skorka, Frank A Petrigliano, and Denis Evseenko. 2022. “A New Mouse Model of Post-Traumatic Joint Injury Allows to Identify the Contribution of Gli1+ Mesenchymal Progenitors in Arthrofibrosis and Acquired Heterotopic Endochondral Ossification.” Frontiers in Cell and Developmental Biology 10 (August): 954028. doi:10.3389/fcell.2022.954028.