Transforming Growth Factor-β Receptor-Mediated, p38 Mitogen-Activated Protein Kinase-Dependent Signaling Drives Enhanced Myofibroblast Differentiation during Skin Wound Healing in Mice Lacking Hyaluronan Synthases 1 and 3.

MLA

Wang, Yan, et al. “Transforming Growth Factor-β Receptor-Mediated, P38 Mitogen-Activated Protein Kinase-Dependent Signaling Drives Enhanced Myofibroblast Differentiation during Skin Wound Healing in Mice Lacking Hyaluronan Synthases 1 and 3.” The American Journal of Pathology, vol. 192, no. 12, Dec. 2022, pp. 1683–98. EBSCOhost, https://doi.org/10.1016/j.ajpath.2022.08.003.

APA

Wang, Y., Mack, J. A., Hascall, V. C., & Maytin, E. V. (2022). Transforming Growth Factor-β Receptor-Mediated, p38 Mitogen-Activated Protein Kinase-Dependent Signaling Drives Enhanced Myofibroblast Differentiation during Skin Wound Healing in Mice Lacking Hyaluronan Synthases 1 and 3. The American Journal of Pathology, 192(12), 1683–1698. https://doi.org/10.1016/j.ajpath.2022.08.003

Chicago

Wang, Yan, Judith A Mack, Vincent C Hascall, and Edward V Maytin. 2022. “Transforming Growth Factor-β Receptor-Mediated, P38 Mitogen-Activated Protein Kinase-Dependent Signaling Drives Enhanced Myofibroblast Differentiation during Skin Wound Healing in Mice Lacking Hyaluronan Synthases 1 and 3.” The American Journal of Pathology 192 (12): 1683–98. doi:10.1016/j.ajpath.2022.08.003.