Cite
Defective fractalkine-CX3CR1 signaling aggravates neuroinflammation and affects recovery from cuprizone-induced demyelination.
MLA
Mendiola, Andrew S., et al. “Defective Fractalkine-CX3CR1 Signaling Aggravates Neuroinflammation and Affects Recovery from Cuprizone-Induced Demyelination.” Journal of Neurochemistry, vol. 162, no. 5, Sept. 2022, pp. 430–43. EBSCOhost, https://doi.org/10.1111/jnc.15616.
APA
Mendiola, A. S., Church, K. A., Cardona, S. M., Vanegas, D., Garcia, S. A., Macklin, W., Lira, S. A., Ransohoff, R. M., Kokovay, E., Lin, C.-H. A., & Cardona, A. E. (2022). Defective fractalkine-CX3CR1 signaling aggravates neuroinflammation and affects recovery from cuprizone-induced demyelination. Journal of Neurochemistry, 162(5), 430–443. https://doi.org/10.1111/jnc.15616
Chicago
Mendiola, Andrew S, Kaira A Church, Sandra M Cardona, Difernando Vanegas, Shannon A Garcia, Wendy Macklin, Sergio A Lira, et al. 2022. “Defective Fractalkine-CX3CR1 Signaling Aggravates Neuroinflammation and Affects Recovery from Cuprizone-Induced Demyelination.” Journal of Neurochemistry 162 (5): 430–43. doi:10.1111/jnc.15616.