Defective dimerization of FoF1-ATP synthase secondary to glycation favors mitochondrial energy deficiency in cardiomyocytes during aging.

MLA

Bou-Teen, Diana, et al. “Defective Dimerization of FoF1-ATP Synthase Secondary to Glycation Favors Mitochondrial Energy Deficiency in Cardiomyocytes during Aging.” Aging Cell, vol. 21, no. 3, Mar. 2022, p. e13564. EBSCOhost, https://doi.org/10.1111/acel.13564.

APA

Bou-Teen, D., Fernandez-Sanz, C., Miro-Casas, E., Nichtova, Z., Bonzon-Kulichenko, E., Casós, K., Inserte, J., Rodriguez-Sinovas, A., Benito, B., Sheu, S.-S., Vázquez, J., Ferreira-González, I., & Ruiz-Meana, M. (2022). Defective dimerization of FoF1-ATP synthase secondary to glycation favors mitochondrial energy deficiency in cardiomyocytes during aging. Aging Cell, 21(3), e13564. https://doi.org/10.1111/acel.13564

Chicago

Bou-Teen, Diana, Celia Fernandez-Sanz, Elisabet Miro-Casas, Zuzana Nichtova, Elena Bonzon-Kulichenko, Kelly Casós, Javier Inserte, et al. 2022. “Defective Dimerization of FoF1-ATP Synthase Secondary to Glycation Favors Mitochondrial Energy Deficiency in Cardiomyocytes during Aging.” Aging Cell 21 (3): e13564. doi:10.1111/acel.13564.