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Loss of SELENOF Induces the Transformed Phenotype in Human Immortalized Prostate Epithelial Cells.
- Source :
-
International journal of molecular sciences [Int J Mol Sci] 2021 Nov 07; Vol. 22 (21). Date of Electronic Publication: 2021 Nov 07. - Publication Year :
- 2021
-
Abstract
- SELENOF is a member of the class of selenoproteins in which the amino acid selenocysteine is co-translationally inserted into the elongating peptide in response to an in-frame UGA codon located in the 3'-untranslated (3'-UTR) region of the SELENOF mRNA. Polymorphisms in the 3'-UTR are associated with an increased risk of dying from prostate cancer and these variations are functional and 10 times more frequent in the genomes of African American men. SELENOF is dramatically reduced in prostate cancer compared to benign adjacent regions. Using a prostate cancer tissue microarray, it was previously established that the reduction of SELENOF in the cancers from African American men was significantly greater than in cancers from Caucasian men. When SELENOF levels in human prostate immortalized epithelial cells were reduced with an shRNA construct, those cells acquired the ability to grow in soft agar, increased the ability to migrate in a scratch assay and acquired features of energy metabolism associated with prostate cancer. These results support a role of SELENOF loss in prostate cancer progression and further indicate that SELENOF loss and genotype may contribute to the disparity in prostate cancer mortality experienced by African American men.
- Subjects :
- Adult
Aged
Case-Control Studies
Cell Line, Transformed
Cells, Cultured
Epithelial Cells metabolism
Genotype
Humans
Male
Middle Aged
Phenotype
Prostate metabolism
Prostatic Neoplasms genetics
Prostatic Neoplasms metabolism
Prostatic Neoplasms pathology
Cell Transformation, Neoplastic genetics
Epithelial Cells pathology
Prostate pathology
Selenoproteins genetics
Subjects
Details
- Language :
- English
- ISSN :
- 1422-0067
- Volume :
- 22
- Issue :
- 21
- Database :
- MEDLINE
- Journal :
- International journal of molecular sciences
- Publication Type :
- Academic Journal
- Accession number :
- 34769469
- Full Text :
- https://doi.org/10.3390/ijms222112040