Ivacaftor restores delayed mucociliary transport caused by Pseudomonas aeruginosa-induced acquired cystic fibrosis transmembrane conductance regulator dysfunction in rabbit nasal epithelia.

MLA

Cho, Do-Yeon, et al. “Ivacaftor Restores Delayed Mucociliary Transport Caused by Pseudomonas Aeruginosa-Induced Acquired Cystic Fibrosis Transmembrane Conductance Regulator Dysfunction in Rabbit Nasal Epithelia.” International Forum of Allergy & Rhinology, vol. 12, no. 5, May 2022, pp. 690–98. EBSCOhost, https://doi.org/10.1002/alr.22907.

APA

Cho, D.-Y., Zhang, S., Skinner, D. F., Lim, D. J., Banks, C., Grayson, J. W., Tearney, G. J., Rowe, S. M., & Woodworth, B. A. (2022). Ivacaftor restores delayed mucociliary transport caused by Pseudomonas aeruginosa-induced acquired cystic fibrosis transmembrane conductance regulator dysfunction in rabbit nasal epithelia. International Forum of Allergy & Rhinology, 12(5), 690–698. https://doi.org/10.1002/alr.22907

Chicago

Cho, Do-Yeon, Shaoyan Zhang, Daniel F Skinner, Dong Jin Lim, Catherine Banks, Jessica W Grayson, Guillermo J Tearney, Steven M Rowe, and Bradford A Woodworth. 2022. “Ivacaftor Restores Delayed Mucociliary Transport Caused by Pseudomonas Aeruginosa-Induced Acquired Cystic Fibrosis Transmembrane Conductance Regulator Dysfunction in Rabbit Nasal Epithelia.” International Forum of Allergy & Rhinology 12 (5): 690–98. doi:10.1002/alr.22907.