Cite
FGFR1 overexpression in non-small cell lung cancer is mediated by genetic and epigenetic mechanisms and is a determinant of FGFR1 inhibitor response.
MLA
Bogatyrova, Olga, et al. “FGFR1 Overexpression in Non-Small Cell Lung Cancer Is Mediated by Genetic and Epigenetic Mechanisms and Is a Determinant of FGFR1 Inhibitor Response.” European Journal of Cancer (Oxford, England : 1990), vol. 151, July 2021, pp. 136–49. EBSCOhost, https://doi.org/10.1016/j.ejca.2021.04.005.
APA
Bogatyrova, O., Mattsson, J. S. M., Ross, E. M., Sanderson, M. P., Backman, M., Botling, J., Brunnström, H., Kurppa, P., La Fleur, L., Strell, C., Wilm, C., Zimmermann, A., Esdar, C., & Micke, P. (2021). FGFR1 overexpression in non-small cell lung cancer is mediated by genetic and epigenetic mechanisms and is a determinant of FGFR1 inhibitor response. European Journal of Cancer (Oxford, England : 1990), 151, 136–149. https://doi.org/10.1016/j.ejca.2021.04.005
Chicago
Bogatyrova, Olga, Johanna S M Mattsson, Edith M Ross, Michael P Sanderson, Max Backman, Johan Botling, Hans Brunnström, et al. 2021. “FGFR1 Overexpression in Non-Small Cell Lung Cancer Is Mediated by Genetic and Epigenetic Mechanisms and Is a Determinant of FGFR1 Inhibitor Response.” European Journal of Cancer (Oxford, England : 1990) 151 (July): 136–49. doi:10.1016/j.ejca.2021.04.005.