Glucotoxicity-induced suppression of Cox6a2 expression provokes β-cell dysfunction via augmented ROS production.

MLA

Nagai, Yasuki, et al. “Glucotoxicity-Induced Suppression of Cox6a2 Expression Provokes β-Cell Dysfunction via Augmented ROS Production.” Biochemical and Biophysical Research Communications, vol. 556, June 2021, pp. 134–41. EBSCOhost, https://doi.org/10.1016/j.bbrc.2021.03.148.

APA

Nagai, Y., Matsuoka, T.-A., Shimo, N., Miyatsuka, T., Miyazaki, S., Tashiro, F., Miyazaki, J.-I., Katakami, N., & Shimomura, I. (2021). Glucotoxicity-induced suppression of Cox6a2 expression provokes β-cell dysfunction via augmented ROS production. Biochemical and Biophysical Research Communications, 556, 134–141. https://doi.org/10.1016/j.bbrc.2021.03.148

Chicago

Nagai, Yasuki, Taka-Aki Matsuoka, Naoki Shimo, Takeshi Miyatsuka, Satsuki Miyazaki, Fumi Tashiro, Jun-Ichi Miyazaki, Naoto Katakami, and Iichiro Shimomura. 2021. “Glucotoxicity-Induced Suppression of Cox6a2 Expression Provokes β-Cell Dysfunction via Augmented ROS Production.” Biochemical and Biophysical Research Communications 556 (June): 134–41. doi:10.1016/j.bbrc.2021.03.148.