Synergistic Utilization of Necrostatin-1 and Z-VAD-FMK Efficiently Promotes the Survival of Compression-Induced Nucleus Pulposus Cells via Alleviating Mitochondrial Dysfunction.

MLA

Chen, Songfeng, et al. “Synergistic Utilization of Necrostatin-1 and Z-VAD-FMK Efficiently Promotes the Survival of Compression-Induced Nucleus Pulposus Cells via Alleviating Mitochondrial Dysfunction.” BioMed Research International, vol. 2020, Dec. 2020, p. 6976317. EBSCOhost, https://doi.org/10.1155/2020/6976317.

APA

Chen, S., Tian, Q., Shang, C., Yang, L., Wei, N., Shang, G., Ji, Y., Kou, H., Lu, S., & Liu, H. (2020). Synergistic Utilization of Necrostatin-1 and Z-VAD-FMK Efficiently Promotes the Survival of Compression-Induced Nucleus Pulposus Cells via Alleviating Mitochondrial Dysfunction. BioMed Research International, 2020, 6976317. https://doi.org/10.1155/2020/6976317

Chicago

Chen, Songfeng, Qing Tian, Chunfeng Shang, Lin Yang, Na Wei, Guowei Shang, Yanhui Ji, Hongwei Kou, Shitao Lu, and Hongjian Liu. 2020. “Synergistic Utilization of Necrostatin-1 and Z-VAD-FMK Efficiently Promotes the Survival of Compression-Induced Nucleus Pulposus Cells via Alleviating Mitochondrial Dysfunction.” BioMed Research International 2020 (December): 6976317. doi:10.1155/2020/6976317.