Cite
20-HETE synthesis inhibition attenuates traumatic brain injury-induced mitochondrial dysfunction and neuronal apoptosis via the SIRT1/PGC-1α pathway: A translational study.
MLA
Cui, Wenxing, et al. “20-HETE Synthesis Inhibition Attenuates Traumatic Brain Injury-Induced Mitochondrial Dysfunction and Neuronal Apoptosis via the SIRT1/PGC-1α Pathway: A Translational Study.” Cell Proliferation, vol. 54, no. 2, Feb. 2021, p. e12964. EBSCOhost, https://doi.org/10.1111/cpr.12964.
APA
Cui, W., Wu, X., Shi, Y., Guo, W., Luo, J., Liu, H., Zheng, L., Du, Y., Wang, P., Wang, Q., Feng, D., Ge, S., & Qu, Y. (2021). 20-HETE synthesis inhibition attenuates traumatic brain injury-induced mitochondrial dysfunction and neuronal apoptosis via the SIRT1/PGC-1α pathway: A translational study. Cell Proliferation, 54(2), e12964. https://doi.org/10.1111/cpr.12964
Chicago
Cui, Wenxing, Xun Wu, Yingwu Shi, Wei Guo, Jianing Luo, Haixiao Liu, Longlong Zheng, et al. 2021. “20-HETE Synthesis Inhibition Attenuates Traumatic Brain Injury-Induced Mitochondrial Dysfunction and Neuronal Apoptosis via the SIRT1/PGC-1α Pathway: A Translational Study.” Cell Proliferation 54 (2): e12964. doi:10.1111/cpr.12964.