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Misunderstandings about developmental dyslexia: a historical overview.

Authors :
Smirni P
Vetri L
Misuraca E
Cappadonna M
Operto FF
Pastorino GMG
Marotta R
Source :
Pediatric reports [Pediatr Rep] 2020 Jun 26; Vol. 12 (2), pp. 8505. Date of Electronic Publication: 2020 Jun 26 (Print Publication: 2020).
Publication Year :
2020

Abstract

Developmental dyslexia is a reading disorder unrelated to intellectual disability, inadequate teaching systems or poor motivation for schooling. The first attempts to understand such difficulty of learning to read, connected the problem to a primary 'visual defect'. Since then, several models have been developed. In the last decades, autopsy and histopathological studies on the brain of developmental dyslexics provided neuroanatomical evidence of structural and morphological differences between the normal and dyslexic brains. Furthermore, neuroimaging studies allowed to understand the neural systems of reading and dyslexia. According to more recent studies, developmental dyslexia appears as a language-related neurodevelopmental disorder with a deficit in phonological decoding and visuospatial organization of the language code. Therefore, dyslexia is viewed as a multicomponential and complex disorder. Consequently, rehabilitation should be aimed at both the recovery of linguistic decoding processes and the improvement of visuo-spatial and attentional processes. This brief overview should be a valuable tool for a deeper understanding of dyslexic disorder. Literature searches in Medline, PsycINFO, EMBASE, Scopus, PubMed, Web of Science identified one hundred articles focusing attention on how this disorder has been considered over the years.<br />Competing Interests: Conflict of interest: The authors declare no potential conflict of interest.<br /> (©Copyright: the Author(s).)

Details

Language :
English
ISSN :
2036-749X
Volume :
12
Issue :
2
Database :
MEDLINE
Journal :
Pediatric reports
Publication Type :
Academic Journal
Accession number :
32922710
Full Text :
https://doi.org/10.4081/pr.2020.8505