Cite
Dioscin Attenuates Interleukin 1β (IL-1β)-Induced Catabolism and Apoptosis via Modulating the Toll-Like Receptor 4 (TLR4)/Nuclear Factor kappa B (NF-κB) Signaling in Human Nucleus Pulposus Cells.
MLA
Wang, Longhui, et al. “Dioscin Attenuates Interleukin 1β (IL-1β)-Induced Catabolism and Apoptosis via Modulating the Toll-Like Receptor 4 (TLR4)/Nuclear Factor Kappa B (NF-ΚB) Signaling in Human Nucleus Pulposus Cells.” Medical Science Monitor : International Medical Journal of Experimental and Clinical Research, vol. 26, Aug. 2020, p. e923386. EBSCOhost, https://doi.org/10.12659/MSM.923386.
APA
Wang, L., Gu, Y., Zhao, H., Chen, R., Chen, W., Qi, H., & Gao, W. (2020). Dioscin Attenuates Interleukin 1β (IL-1β)-Induced Catabolism and Apoptosis via Modulating the Toll-Like Receptor 4 (TLR4)/Nuclear Factor kappa B (NF-κB) Signaling in Human Nucleus Pulposus Cells. Medical Science Monitor : International Medical Journal of Experimental and Clinical Research, 26, e923386. https://doi.org/10.12659/MSM.923386
Chicago
Wang, Longhui, Yuntao Gu, Hai Zhao, Rong Chen, Wensheng Chen, Hao Qi, and Weisong Gao. 2020. “Dioscin Attenuates Interleukin 1β (IL-1β)-Induced Catabolism and Apoptosis via Modulating the Toll-Like Receptor 4 (TLR4)/Nuclear Factor Kappa B (NF-ΚB) Signaling in Human Nucleus Pulposus Cells.” Medical Science Monitor : International Medical Journal of Experimental and Clinical Research 26 (August): e923386. doi:10.12659/MSM.923386.