Cholinergic-like neurons carrying PSEN1 E280A mutation from familial Alzheimer's disease reveal intraneuronal sAPPβ fragments accumulation, hyperphosphorylation of TAU, oxidative stress, apoptosis and Ca2+ dysregulation: Therapeutic implications.

MLA

Soto-Mercado, Viviana, et al. “Cholinergic-like Neurons Carrying PSEN1 E280A Mutation from Familial Alzheimer’s Disease Reveal Intraneuronal SAPPβ Fragments Accumulation, Hyperphosphorylation of TAU, Oxidative Stress, Apoptosis and Ca2+ Dysregulation: Therapeutic Implications.” PloS One, vol. 15, no. 5, May 2020, p. e0221669. EBSCOhost, https://doi.org/10.1371/journal.pone.0221669.

APA

Soto-Mercado, V., Mendivil-Perez, M., Velez-Pardo, C., Lopera, F., & Jimenez-Del-Rio, M. (2020). Cholinergic-like neurons carrying PSEN1 E280A mutation from familial Alzheimer’s disease reveal intraneuronal sAPPβ fragments accumulation, hyperphosphorylation of TAU, oxidative stress, apoptosis and Ca2+ dysregulation: Therapeutic implications. PloS One, 15(5), e0221669. https://doi.org/10.1371/journal.pone.0221669

Chicago

Soto-Mercado, Viviana, Miguel Mendivil-Perez, Carlos Velez-Pardo, Francisco Lopera, and Marlene Jimenez-Del-Rio. 2020. “Cholinergic-like Neurons Carrying PSEN1 E280A Mutation from Familial Alzheimer’s Disease Reveal Intraneuronal SAPPβ Fragments Accumulation, Hyperphosphorylation of TAU, Oxidative Stress, Apoptosis and Ca2+ Dysregulation: Therapeutic Implications.” PloS One 15 (5): e0221669. doi:10.1371/journal.pone.0221669.