An optically active isochroman-2H-chromene conjugate potently suppresses neuronal oxidative injuries associated with the PI3K/Akt and MAPK signaling pathways.

MLA

Tao, Ling-Xue, et al. “An Optically Active Isochroman-2H-Chromene Conjugate Potently Suppresses Neuronal Oxidative Injuries Associated with the PI3K/Akt and MAPK Signaling Pathways.” Acta Pharmacologica Sinica, vol. 42, no. 1, Jan. 2021, pp. 36–44. EBSCOhost, https://doi.org/10.1038/s41401-020-0391-9.

APA

Tao, L.-X., Ji, S.-S., Szalóki, D., Kovács, T., Mándi, A., Antus, S., Ding, X., Kurtán, T., & Zhang, H.-Y. (2021). An optically active isochroman-2H-chromene conjugate potently suppresses neuronal oxidative injuries associated with the PI3K/Akt and MAPK signaling pathways. Acta Pharmacologica Sinica, 42(1), 36–44. https://doi.org/10.1038/s41401-020-0391-9

Chicago

Tao, Ling-Xue, Sha-Sha Ji, Dóra Szalóki, Tibor Kovács, Attila Mándi, Sándor Antus, Xun Ding, Tibor Kurtán, and Hai-Yan Zhang. 2021. “An Optically Active Isochroman-2H-Chromene Conjugate Potently Suppresses Neuronal Oxidative Injuries Associated with the PI3K/Akt and MAPK Signaling Pathways.” Acta Pharmacologica Sinica 42 (1): 36–44. doi:10.1038/s41401-020-0391-9.