Amorphous SiO2 nanoparticles promote cardiac dysfunction via the opening of the mitochondrial permeability transition pore in rat heart and human cardiomyocytes.

MLA

Lozano, Omar, et al. “Amorphous SiO2 Nanoparticles Promote Cardiac Dysfunction via the Opening of the Mitochondrial Permeability Transition Pore in Rat Heart and Human Cardiomyocytes.” Particle and Fibre Toxicology, vol. 17, no. 1, May 2020, p. 15. EBSCOhost, https://doi.org/10.1186/s12989-020-00346-2.

APA

Lozano, O., Silva-Platas, C., Chapoy-Villanueva, H., Pérez, B. E., Lees, J. G., Ramachandra, C. J. A., Contreras-Torres, F. F., Lázaro-Alfaro, A., Luna-Figueroa, E., Bernal-Ramírez, J., Gordillo-Galeano, A., Benitez, A., Oropeza-Almazán, Y., Castillo, E. C., Koh, P. L., Hausenloy, D. J., Lim, S. Y., & García-Rivas, G. (2020). Amorphous SiO2 nanoparticles promote cardiac dysfunction via the opening of the mitochondrial permeability transition pore in rat heart and human cardiomyocytes. Particle and Fibre Toxicology, 17(1), 15. https://doi.org/10.1186/s12989-020-00346-2

Chicago

Lozano, Omar, Christian Silva-Platas, Héctor Chapoy-Villanueva, Baruc E Pérez, Jarmon G Lees, Chrishan J A Ramachandra, Flavio F Contreras-Torres, et al. 2020. “Amorphous SiO2 Nanoparticles Promote Cardiac Dysfunction via the Opening of the Mitochondrial Permeability Transition Pore in Rat Heart and Human Cardiomyocytes.” Particle and Fibre Toxicology 17 (1): 15. doi:10.1186/s12989-020-00346-2.