Novel IQCE variations confirm its role in postaxial polydactyly and cause ciliary defect phenotype in zebrafish.

MLA

Estrada-Cuzcano, Alejandro, et al. “Novel IQCE Variations Confirm Its Role in Postaxial Polydactyly and Cause Ciliary Defect Phenotype in Zebrafish.” Human Mutation, vol. 41, no. 1, Jan. 2020, pp. 240–54. EBSCOhost, https://doi.org/10.1002/humu.23924.

APA

Estrada-Cuzcano, A., Etard, C., Delvallée, C., Stoetzel, C., Schaefer, E., Scheidecker, S., Geoffroy, V., Schneider, A., Studer, F., Mattioli, F., Chennen, K., Sigaudy, S., Plassard, D., Poch, O., Piton, A., Strahle, U., Muller, J., & Dollfus, H. (2020). Novel IQCE variations confirm its role in postaxial polydactyly and cause ciliary defect phenotype in zebrafish. Human Mutation, 41(1), 240–254. https://doi.org/10.1002/humu.23924

Chicago

Estrada-Cuzcano, Alejandro, Christelle Etard, Clarisse Delvallée, Corinne Stoetzel, Elise Schaefer, Sophie Scheidecker, Véronique Geoffroy, et al. 2020. “Novel IQCE Variations Confirm Its Role in Postaxial Polydactyly and Cause Ciliary Defect Phenotype in Zebrafish.” Human Mutation 41 (1): 240–54. doi:10.1002/humu.23924.