Exosomal CagA derived from Helicobacter pylori-infected gastric epithelial cells induces macrophage foam cell formation and promotes atherosclerosis.

MLA

Yang, Shuai, et al. “Exosomal CagA Derived from Helicobacter Pylori-Infected Gastric Epithelial Cells Induces Macrophage Foam Cell Formation and Promotes Atherosclerosis.” Journal of Molecular and Cellular Cardiology, vol. 135, Oct. 2019, pp. 40–51. EBSCOhost, https://doi.org/10.1016/j.yjmcc.2019.07.011.

APA

Yang, S., Xia, Y.-P., Luo, X.-Y., Chen, S.-L., Li, B.-W., Ye, Z.-M., Chen, S.-C., Mao, L., Jin, H.-J., Li, Y.-N., & Hu, B. (2019). Exosomal CagA derived from Helicobacter pylori-infected gastric epithelial cells induces macrophage foam cell formation and promotes atherosclerosis. Journal of Molecular and Cellular Cardiology, 135, 40–51. https://doi.org/10.1016/j.yjmcc.2019.07.011

Chicago

Yang, Shuai, Yuan-Peng Xia, Xue-Ying Luo, Shao-Li Chen, Bo-Wei Li, Zi-Ming Ye, Sheng-Cai Chen, et al. 2019. “Exosomal CagA Derived from Helicobacter Pylori-Infected Gastric Epithelial Cells Induces Macrophage Foam Cell Formation and Promotes Atherosclerosis.” Journal of Molecular and Cellular Cardiology 135 (October): 40–51. doi:10.1016/j.yjmcc.2019.07.011.