An Epilepsy-Associated KCNT1 Mutation Enhances Excitability of Human iPSC-Derived Neurons by Increasing Slack K Na Currents.

MLA

Quraishi, Imran H., et al. “An Epilepsy-Associated KCNT1 Mutation Enhances Excitability of Human IPSC-Derived Neurons by Increasing Slack K Na Currents.” The Journal of Neuroscience : The Official Journal of the Society for Neuroscience, vol. 39, no. 37, Sept. 2019, pp. 7438–49. EBSCOhost, https://doi.org/10.1523/JNEUROSCI.1628-18.2019.

APA

Quraishi, I. H., Stern, S., Mangan, K. P., Zhang, Y., Ali, S. R., Mercier, M. R., Marchetto, M. C., McLachlan, M. J., Jones, E. M., Gage, F. H., & Kaczmarek, L. K. (2019). An Epilepsy-Associated KCNT1 Mutation Enhances Excitability of Human iPSC-Derived Neurons by Increasing Slack K Na Currents. The Journal of Neuroscience : The Official Journal of the Society for Neuroscience, 39(37), 7438–7449. https://doi.org/10.1523/JNEUROSCI.1628-18.2019

Chicago

Quraishi, Imran H, Shani Stern, Kile P Mangan, Yalan Zhang, Syed R Ali, Michael R Mercier, Maria C Marchetto, et al. 2019. “An Epilepsy-Associated KCNT1 Mutation Enhances Excitability of Human IPSC-Derived Neurons by Increasing Slack K Na Currents.” The Journal of Neuroscience : The Official Journal of the Society for Neuroscience 39 (37): 7438–49. doi:10.1523/JNEUROSCI.1628-18.2019.