GSK3β-mediated Keap1-independent regulation of Nrf2 antioxidant response: A molecular rheostat of acute kidney injury to chronic kidney disease transition.

MLA

Lu, Minglei, et al. “GSK3β-Mediated Keap1-Independent Regulation of Nrf2 Antioxidant Response: A Molecular Rheostat of Acute Kidney Injury to Chronic Kidney Disease Transition.” Redox Biology, vol. 26, Sept. 2019, p. 101275. EBSCOhost, https://doi.org/10.1016/j.redox.2019.101275.

APA

Lu, M., Wang, P., Qiao, Y., Jiang, C., Ge, Y., Flickinger, B., Malhotra, D. K., Dworkin, L. D., Liu, Z., & Gong, R. (2019). GSK3β-mediated Keap1-independent regulation of Nrf2 antioxidant response: A molecular rheostat of acute kidney injury to chronic kidney disease transition. Redox Biology, 26, 101275. https://doi.org/10.1016/j.redox.2019.101275

Chicago

Lu, Minglei, Pei Wang, Yingjin Qiao, Chunming Jiang, Yan Ge, Bryce Flickinger, Deepak K Malhotra, Lance D Dworkin, Zhangsuo Liu, and Rujun Gong. 2019. “GSK3β-Mediated Keap1-Independent Regulation of Nrf2 Antioxidant Response: A Molecular Rheostat of Acute Kidney Injury to Chronic Kidney Disease Transition.” Redox Biology 26 (September): 101275. doi:10.1016/j.redox.2019.101275.