Upregulation of endogenous thrombopoietin receptor (MPL) with in vivo passage of calreticulin (CALR) mutant Ba/F3 cells, highlighting MPL as the requisite cytokine receptor for CALR mediated transformation.

MLA

Brooks, Stefan A., et al. “Upregulation of Endogenous Thrombopoietin Receptor (MPL) with in Vivo Passage of Calreticulin (CALR) Mutant Ba/F3 Cells, Highlighting MPL as the Requisite Cytokine Receptor for CALR Mediated Transformation.” Leukemia Research, vol. 82, July 2019, pp. 11–14. EBSCOhost, https://doi.org/10.1016/j.leukres.2019.05.003.

APA

Brooks, S. A., Kim, D. M., Morse, S. J., Nguyen, Q. H., Craver, B. M., Lai, H. Y., & Fleischman, A. G. (2019). Upregulation of endogenous thrombopoietin receptor (MPL) with in vivo passage of calreticulin (CALR) mutant Ba/F3 cells, highlighting MPL as the requisite cytokine receptor for CALR mediated transformation. Leukemia Research, 82, 11–14. https://doi.org/10.1016/j.leukres.2019.05.003

Chicago

Brooks, Stefan A, Daniel M Kim, Sarah J Morse, Quy H Nguyen, Brianna M Craver, Hew Yeng Lai, and Angela G Fleischman. 2019. “Upregulation of Endogenous Thrombopoietin Receptor (MPL) with in Vivo Passage of Calreticulin (CALR) Mutant Ba/F3 Cells, Highlighting MPL as the Requisite Cytokine Receptor for CALR Mediated Transformation.” Leukemia Research 82 (July): 11–14. doi:10.1016/j.leukres.2019.05.003.